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emg amplifier labchart 16-channel physiological signal recording and analysis system bioelectric amplifier  (ADInstruments)


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    ADInstruments emg amplifier labchart 16-channel physiological signal recording and analysis system bioelectric amplifier
    ( A ) Flowchart of experiment one. All ten-day-old SD rats except for control rats received iodoacetamide (IA) daily for 6 days and housed normally to grow until adulthood of 8 weeks old. Surgical implantations for ECG and <t>EMG</t> were performed at the age of 8 weeks and then recovered for 1 <t>week.</t> <t>Physiological</t> measurements were made at the 9th week and after the 11th week. Wire implantation for aVNS or sham-aVNS were performed at the age of 8 weeks and then recovered for 1 week at each group. The treatment (aVNS or sham-aVNS) was conducted for 2 weeks (9th week to 11th week). Only the EMG was performed after the 9th week and 10th week. ( B ) Flowchart of experiment two. All ten-day-old SD rats received the same IA treatment. Subphrenic vagotomy were performed at the age of 8 weeks and then recovered for 1 week. aVNS was conducted as experiment one. Physiological measurements were made after the 11th week. ( C ) aVNS schematic diagram.
    Emg Amplifier Labchart 16 Channel Physiological Signal Recording And Analysis System Bioelectric Amplifier, supplied by ADInstruments, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/labchart+16-channel+physiological+signal+recording+and+analysis+system+bioelectric+amplifier/16+channel+power+lab/pmc09954117-230-21-33
    Average 90 stars, based on 1 article reviews
    emg amplifier labchart 16-channel physiological signal recording and analysis system bioelectric amplifier - by Bioz Stars, 2026-09
    90/100 stars

    Images

    1) Product Images from "Auricular Vagus Nerve Stimulation Improves Visceral Hypersensitivity and Gastric Motility and Depression-like Behaviors via Vago-Vagal Pathway in a Rat Model of Functional Dyspepsia"

    Article Title: Auricular Vagus Nerve Stimulation Improves Visceral Hypersensitivity and Gastric Motility and Depression-like Behaviors via Vago-Vagal Pathway in a Rat Model of Functional Dyspepsia

    Journal: Brain Sciences

    doi: 10.3390/brainsci13020253

    ( A ) Flowchart of experiment one. All ten-day-old SD rats except for control rats received iodoacetamide (IA) daily for 6 days and housed normally to grow until adulthood of 8 weeks old. Surgical implantations for ECG and EMG were performed at the age of 8 weeks and then recovered for 1 week. Physiological measurements were made at the 9th week and after the 11th week. Wire implantation for aVNS or sham-aVNS were performed at the age of 8 weeks and then recovered for 1 week at each group. The treatment (aVNS or sham-aVNS) was conducted for 2 weeks (9th week to 11th week). Only the EMG was performed after the 9th week and 10th week. ( B ) Flowchart of experiment two. All ten-day-old SD rats received the same IA treatment. Subphrenic vagotomy were performed at the age of 8 weeks and then recovered for 1 week. aVNS was conducted as experiment one. Physiological measurements were made after the 11th week. ( C ) aVNS schematic diagram.
    Figure Legend Snippet: ( A ) Flowchart of experiment one. All ten-day-old SD rats except for control rats received iodoacetamide (IA) daily for 6 days and housed normally to grow until adulthood of 8 weeks old. Surgical implantations for ECG and EMG were performed at the age of 8 weeks and then recovered for 1 week. Physiological measurements were made at the 9th week and after the 11th week. Wire implantation for aVNS or sham-aVNS were performed at the age of 8 weeks and then recovered for 1 week at each group. The treatment (aVNS or sham-aVNS) was conducted for 2 weeks (9th week to 11th week). Only the EMG was performed after the 9th week and 10th week. ( B ) Flowchart of experiment two. All ten-day-old SD rats received the same IA treatment. Subphrenic vagotomy were performed at the age of 8 weeks and then recovered for 1 week. aVNS was conducted as experiment one. Physiological measurements were made after the 11th week. ( C ) aVNS schematic diagram.

    Techniques Used: Control

    Assessment of rat model. ( a ) Different EMG waveforms changes response to gastric distension (GD) of 20, 40, 60 and 80 mmHg in control rats and IA-treated rats. ( b ) Gastric emptying results in control rats and IA-treated rats. The index was detected from 12 sacrificed rats after the 9th week (6 rats were randomly picked in control group and IA-treated group, respectively). The independent sample t-test was used. ( c ) EMG change ratio response to GD in all rats when rat model was induced. ( d ) The open field test (OFT) results when rat model was induced in all rats. All indexes were tested after the 9th week and before aVNS or sham-aVNS. One-way ANOVA was used, and the LSD method was used for post hoc tests in EMG change ratio and OFT. EMG: electromyography. * p < 0.05, ** p < 0.01, **** p < 0.0001. C: control group; IA: IA-treated group. n = 6 per group. Data were presented as means ± standard deviation.
    Figure Legend Snippet: Assessment of rat model. ( a ) Different EMG waveforms changes response to gastric distension (GD) of 20, 40, 60 and 80 mmHg in control rats and IA-treated rats. ( b ) Gastric emptying results in control rats and IA-treated rats. The index was detected from 12 sacrificed rats after the 9th week (6 rats were randomly picked in control group and IA-treated group, respectively). The independent sample t-test was used. ( c ) EMG change ratio response to GD in all rats when rat model was induced. ( d ) The open field test (OFT) results when rat model was induced in all rats. All indexes were tested after the 9th week and before aVNS or sham-aVNS. One-way ANOVA was used, and the LSD method was used for post hoc tests in EMG change ratio and OFT. EMG: electromyography. * p < 0.05, ** p < 0.01, **** p < 0.0001. C: control group; IA: IA-treated group. n = 6 per group. Data were presented as means ± standard deviation.

    Techniques Used: Control, Standard Deviation

    Effects of the aVNS. ( A ) EMG change ratio results after one-week intervention in all rats. ( B ) Comparison results of EMG change ratio before and after aVNS. ( C ) Comparison results of EMG change ratio before and after sham-aVNS. ( D ) Gastric emptying after two-week intervention in all rats. ( E , F ) OFT results after two-week intervention in all rats. ( G ) Comparison results of horizontal scores before and after aVNS. ( H ) Comparison results of vertical scores before and after sham-aVNS. A paired t-test was adopted for comparison in ( B , C , G , H ). One-way ANOVA was used, and the LSD method was used for post hoc tests in ( A , D , E , F ) (except for 80 mmHg in A Tamhane’s T2(M) method was used). EMG: electromyography. * p < 0.05, ** p < 0.01, *** p < 0.001, **** p < 0.0001. C: control group; IA: IA-treated group. n = 6 per group. Data were presented as means ± standard deviation.
    Figure Legend Snippet: Effects of the aVNS. ( A ) EMG change ratio results after one-week intervention in all rats. ( B ) Comparison results of EMG change ratio before and after aVNS. ( C ) Comparison results of EMG change ratio before and after sham-aVNS. ( D ) Gastric emptying after two-week intervention in all rats. ( E , F ) OFT results after two-week intervention in all rats. ( G ) Comparison results of horizontal scores before and after aVNS. ( H ) Comparison results of vertical scores before and after sham-aVNS. A paired t-test was adopted for comparison in ( B , C , G , H ). One-way ANOVA was used, and the LSD method was used for post hoc tests in ( A , D , E , F ) (except for 80 mmHg in A Tamhane’s T2(M) method was used). EMG: electromyography. * p < 0.05, ** p < 0.01, *** p < 0.001, **** p < 0.0001. C: control group; IA: IA-treated group. n = 6 per group. Data were presented as means ± standard deviation.

    Techniques Used: Comparison, Control, Standard Deviation

    Related Articles

    Marker:

    Article Title: Effect and mechanism study on transcutaneous auricular vagus nerve stimulation for functional dyspepsia model rats 经皮耳迷走神经刺激干预功能性消化不良模型大鼠的效应及机制研究
    Article Snippet: Objective: To explore the effect and its possible mechanism of the intervention of transcutaneous auricular vagus nerve stimulation (taVNS) for the functional dyspepsia (FD) model rats.. Methods: Of the 25 male SD rats, 6 rats were set as blank group, the other 19 rats were established to be the functional dyspepsia (FD) model by iodoacetamide intragastric administration, and 18 FD models were successfully established.. The 18 model rats were randomly divided into a model group, a sham-taVNS group, and taVNS group, with 6 rats in each group.

    Injection:

    Article Title: Effect and mechanism study on transcutaneous auricular vagus nerve stimulation for functional dyspepsia model rats 经皮耳迷走神经刺激干预功能性消化不良模型大鼠的效应及机制研究
    Article Snippet: Objective: To explore the effect and its possible mechanism of the intervention of transcutaneous auricular vagus nerve stimulation (taVNS) for the functional dyspepsia (FD) model rats.. Methods: Of the 25 male SD rats, 6 rats were set as blank group, the other 19 rats were established to be the functional dyspepsia (FD) model by iodoacetamide intragastric administration, and 18 FD models were successfully established.. The 18 model rats were randomly divided into a model group, a sham-taVNS group, and taVNS group, with 6 rats in each group.

    Enzyme-linked Immunosorbent Assay:

    Article Title: Effect and mechanism study on transcutaneous auricular vagus nerve stimulation for functional dyspepsia model rats 经皮耳迷走神经刺激干预功能性消化不良模型大鼠的效应及机制研究
    Article Snippet: Objective: To explore the effect and its possible mechanism of the intervention of transcutaneous auricular vagus nerve stimulation (taVNS) for the functional dyspepsia (FD) model rats.. Methods: Of the 25 male SD rats, 6 rats were set as blank group, the other 19 rats were established to be the functional dyspepsia (FD) model by iodoacetamide intragastric administration, and 18 FD models were successfully established.. The 18 model rats were randomly divided into a model group, a sham-taVNS group, and taVNS group, with 6 rats in each group.



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    ( A ) Flowchart of experiment one. All ten-day-old SD rats except for control rats received iodoacetamide (IA) daily for 6 days and housed normally to grow until adulthood of 8 weeks old. Surgical implantations for ECG and <t>EMG</t> were performed at the age of 8 weeks and then recovered for 1 <t>week.</t> <t>Physiological</t> measurements were made at the 9th week and after the 11th week. Wire implantation for aVNS or sham-aVNS were performed at the age of 8 weeks and then recovered for 1 week at each group. The treatment (aVNS or sham-aVNS) was conducted for 2 weeks (9th week to 11th week). Only the EMG was performed after the 9th week and 10th week. ( B ) Flowchart of experiment two. All ten-day-old SD rats received the same IA treatment. Subphrenic vagotomy were performed at the age of 8 weeks and then recovered for 1 week. aVNS was conducted as experiment one. Physiological measurements were made after the 11th week. ( C ) aVNS schematic diagram.
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    Image Search Results


    ( A ) Flowchart of experiment one. All ten-day-old SD rats except for control rats received iodoacetamide (IA) daily for 6 days and housed normally to grow until adulthood of 8 weeks old. Surgical implantations for ECG and EMG were performed at the age of 8 weeks and then recovered for 1 week. Physiological measurements were made at the 9th week and after the 11th week. Wire implantation for aVNS or sham-aVNS were performed at the age of 8 weeks and then recovered for 1 week at each group. The treatment (aVNS or sham-aVNS) was conducted for 2 weeks (9th week to 11th week). Only the EMG was performed after the 9th week and 10th week. ( B ) Flowchart of experiment two. All ten-day-old SD rats received the same IA treatment. Subphrenic vagotomy were performed at the age of 8 weeks and then recovered for 1 week. aVNS was conducted as experiment one. Physiological measurements were made after the 11th week. ( C ) aVNS schematic diagram.

    Journal: Brain Sciences

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    Figure Lengend Snippet: ( A ) Flowchart of experiment one. All ten-day-old SD rats except for control rats received iodoacetamide (IA) daily for 6 days and housed normally to grow until adulthood of 8 weeks old. Surgical implantations for ECG and EMG were performed at the age of 8 weeks and then recovered for 1 week. Physiological measurements were made at the 9th week and after the 11th week. Wire implantation for aVNS or sham-aVNS were performed at the age of 8 weeks and then recovered for 1 week at each group. The treatment (aVNS or sham-aVNS) was conducted for 2 weeks (9th week to 11th week). Only the EMG was performed after the 9th week and 10th week. ( B ) Flowchart of experiment two. All ten-day-old SD rats received the same IA treatment. Subphrenic vagotomy were performed at the age of 8 weeks and then recovered for 1 week. aVNS was conducted as experiment one. Physiological measurements were made after the 11th week. ( C ) aVNS schematic diagram.

    Article Snippet: EMG recording: EMG responses to gastric distension (GD) at different pressures (20, 40, 60 and 80 mmHg) were recorded using an EMG amplifier (Labchart 16-channel physiological signal recording and analysis system bioelectric amplifier, ADINSTRUMENTS, Dunedin, New Zealand).

    Techniques: Control

    Assessment of rat model. ( a ) Different EMG waveforms changes response to gastric distension (GD) of 20, 40, 60 and 80 mmHg in control rats and IA-treated rats. ( b ) Gastric emptying results in control rats and IA-treated rats. The index was detected from 12 sacrificed rats after the 9th week (6 rats were randomly picked in control group and IA-treated group, respectively). The independent sample t-test was used. ( c ) EMG change ratio response to GD in all rats when rat model was induced. ( d ) The open field test (OFT) results when rat model was induced in all rats. All indexes were tested after the 9th week and before aVNS or sham-aVNS. One-way ANOVA was used, and the LSD method was used for post hoc tests in EMG change ratio and OFT. EMG: electromyography. * p < 0.05, ** p < 0.01, **** p < 0.0001. C: control group; IA: IA-treated group. n = 6 per group. Data were presented as means ± standard deviation.

    Journal: Brain Sciences

    Article Title: Auricular Vagus Nerve Stimulation Improves Visceral Hypersensitivity and Gastric Motility and Depression-like Behaviors via Vago-Vagal Pathway in a Rat Model of Functional Dyspepsia

    doi: 10.3390/brainsci13020253

    Figure Lengend Snippet: Assessment of rat model. ( a ) Different EMG waveforms changes response to gastric distension (GD) of 20, 40, 60 and 80 mmHg in control rats and IA-treated rats. ( b ) Gastric emptying results in control rats and IA-treated rats. The index was detected from 12 sacrificed rats after the 9th week (6 rats were randomly picked in control group and IA-treated group, respectively). The independent sample t-test was used. ( c ) EMG change ratio response to GD in all rats when rat model was induced. ( d ) The open field test (OFT) results when rat model was induced in all rats. All indexes were tested after the 9th week and before aVNS or sham-aVNS. One-way ANOVA was used, and the LSD method was used for post hoc tests in EMG change ratio and OFT. EMG: electromyography. * p < 0.05, ** p < 0.01, **** p < 0.0001. C: control group; IA: IA-treated group. n = 6 per group. Data were presented as means ± standard deviation.

    Article Snippet: EMG recording: EMG responses to gastric distension (GD) at different pressures (20, 40, 60 and 80 mmHg) were recorded using an EMG amplifier (Labchart 16-channel physiological signal recording and analysis system bioelectric amplifier, ADINSTRUMENTS, Dunedin, New Zealand).

    Techniques: Control, Standard Deviation

    Effects of the aVNS. ( A ) EMG change ratio results after one-week intervention in all rats. ( B ) Comparison results of EMG change ratio before and after aVNS. ( C ) Comparison results of EMG change ratio before and after sham-aVNS. ( D ) Gastric emptying after two-week intervention in all rats. ( E , F ) OFT results after two-week intervention in all rats. ( G ) Comparison results of horizontal scores before and after aVNS. ( H ) Comparison results of vertical scores before and after sham-aVNS. A paired t-test was adopted for comparison in ( B , C , G , H ). One-way ANOVA was used, and the LSD method was used for post hoc tests in ( A , D , E , F ) (except for 80 mmHg in A Tamhane’s T2(M) method was used). EMG: electromyography. * p < 0.05, ** p < 0.01, *** p < 0.001, **** p < 0.0001. C: control group; IA: IA-treated group. n = 6 per group. Data were presented as means ± standard deviation.

    Journal: Brain Sciences

    Article Title: Auricular Vagus Nerve Stimulation Improves Visceral Hypersensitivity and Gastric Motility and Depression-like Behaviors via Vago-Vagal Pathway in a Rat Model of Functional Dyspepsia

    doi: 10.3390/brainsci13020253

    Figure Lengend Snippet: Effects of the aVNS. ( A ) EMG change ratio results after one-week intervention in all rats. ( B ) Comparison results of EMG change ratio before and after aVNS. ( C ) Comparison results of EMG change ratio before and after sham-aVNS. ( D ) Gastric emptying after two-week intervention in all rats. ( E , F ) OFT results after two-week intervention in all rats. ( G ) Comparison results of horizontal scores before and after aVNS. ( H ) Comparison results of vertical scores before and after sham-aVNS. A paired t-test was adopted for comparison in ( B , C , G , H ). One-way ANOVA was used, and the LSD method was used for post hoc tests in ( A , D , E , F ) (except for 80 mmHg in A Tamhane’s T2(M) method was used). EMG: electromyography. * p < 0.05, ** p < 0.01, *** p < 0.001, **** p < 0.0001. C: control group; IA: IA-treated group. n = 6 per group. Data were presented as means ± standard deviation.

    Article Snippet: EMG recording: EMG responses to gastric distension (GD) at different pressures (20, 40, 60 and 80 mmHg) were recorded using an EMG amplifier (Labchart 16-channel physiological signal recording and analysis system bioelectric amplifier, ADINSTRUMENTS, Dunedin, New Zealand).

    Techniques: Comparison, Control, Standard Deviation